Here are the facts: two people can eat EXACTLY the same number of calories, and have exactly the same amount of activity, yet one person will stay the same or lose weight while the other gains weight.
THIS IS BECAUSE THERE ARE MORE FACTORS AT WORK THAN JUST 'CALORIES IN - CALORIES OUT'.
Actually calories in - calories out does explain the body storing energy as fat or not. The "more factors" is in how efficiently an individual's digestive system extracts calories from the food consumed and how many calories their body burns in daily activities. Of course, however, there are medical conditions that affects one's metabolism for instance someone with very active thrifty genes needs to consume less and/or be more active if they don't want to store fat.
Which has been my entire point. Given exactly the same amount of "calories in" and the same exact amount of physical activity, different people will gain, lose or maintain their weight because their individual bodies process the energy (calories in) differently. How one's body processes those calories is largely genetic and can also be affected by medical conditions, medicines, even stress. There is also growing evidence that the source of the calorie matters - the same exact caloric value of protein is processed different than simple carbs than complex carbs than fats. All of these need to be (at least) acknowledged in a discussion like this, instead of all of the really disgusting fat-shaming several people have engaged in.
But in the real world, the overwhelming majority of overweight people do consume more calories and/or are less active than underweight people.
And here is where I disagree. Are there people who over-consume calories? Of course. I never said there weren't. But it does not do anyone any good to broad-brush everyone as "lazy, no-self-control, fat slobs" just because of their apparent weight - which is exactly what certain people in this thread are doing, and what too many assholes everywhere do.
In the real world, there are hundreds of factors - many of which are beyond an individual's control - that are contributing to
a nearly universal increase in weight (the point the OP article was making before the fat-shamers piped in). Decrease in physical activity is one - but that affects different bodies differently. Increase in sugars and corn-syrups in virtually every processed food we purchase is another - which again affects different bodies differently. A lot of the sugar we consume is within our control - yes. A lot is hidden and many people may not even be aware of it. Examples: barbecue sauces, breads, canned-fruits, crackers, frozen dinners, hot dogs, ketchup, marinades, peanut butter, pickles, salad dressing, soup, vegetables...
vegetables! The stuff that is supposed to be good for us.
And as you agreed above, the food we eat is processed differently by different individual bodies, so what may not affect you at all could cause me to gain weight.
To argue differently would necessitate proposing that something happened in the gene pool of the world's developed nations (but not in third world nations) over the last fifty years.
Not necessarily in the gene pool, though research is showing that genetics plays a bigger role than previously thought.
In the mouse study, the research team determined that mutations in the Mrap2 gene led the animals to eat less initially but still gain about twice as much weight as they normally would. While their appetites returned, these mice continued to gain weight despite being fed the same number of calories as a group of control animals. That led the scientists to figure out that the mice with the mutated gene were simply sequestering fat rather than breaking it down for energy. The mice, like people, possessed two copies of the gene, and mice with even one defective copy experienced significant weight gain, although not as much as those who had two mutated versions of Mrap2.
http://healthland.time.com/2013/07/19/news-genes-idd-in-obesity-how-much-of-weight-is-genetic/
In a very closely controlled, year-long study with post-menopausal women-chosen as subjects because they are more stable, hormonally speaking-researchers divided the participants into an exercise intervention group and a control group.
All the subjects were carefully selected for specific features of their health and lifestyle. Various lab tests were taken, including certain genetic tests. The subjects were all instructed not to change their dietary habits. The control group was assigned a moderate stretching regimen to use over the course of the study, and the intervention group was given a moderate intensity, 45-minute workout for five days a week, initially with supervision. There was detailed tracking, because the researchers were especially interested in identifying distinctions between genetic and environmental factors.
At the end of the study period, the researchers found a distinct variability in the amount of fat lost by some exercisers that could not be explained by their adherence levels.
So the researchers looked to to the genetic data, and indeed, there were two specific genes that stood out related to patterns of fat loss and exercise. Participants with certain combinations of those genes and their variants had significantly different fat loss outcomes.
The complex science at this level can be pretty confusing. Genes are named with different alpha-numeric codes and so are their variant alleles. If you're not careful, you can end up in some real alphabet soup just trying to talk about it, but basically two genes are involved here, the CYP19 and the COMT.
They found that having one CYP19 gene and its variants was not consistently associated with percentage fat, total fat, subcutaneous fat or intra-abdominal fat, that dangerous excess that gathers around our vital organs. But having two such genes correlated to a slightly larger decrease in body mass index, and a significantly larger decrease in total fat and percentage of body fat.
And having a particular genotype of the COMT gene, together with at least one copy of the CYP19 gene, also related to a significantly larger decrease in BMI, total fat and percentage of body fat. There were corresponding intermediate combinations and results that bore out the relationships.
Interestingly, in the exercising group, those with a specific variant of the CYP19 gene lost more fat over the year, but in the non-workout group, those with that same variant gained more fat.
Why and how these genes affect the fat/exercise relationship isn't yet understood, but now that these have been identified as significant, they are the subject of much new, focused study. Other research has since showed that women with that specific CYP19 variant have higher concentrations of the hormones estrone and estradiol, which may make them more resistant to fat loss through exercise.
http://www.bistromd.com/weight-loss/the-truth-about-genetics-and-weight-loss
and so on...
However, you are incorrect to assume that third-world countries do not have obesity rates like the US or other developed countries. In fact, developing nations have about 2/3's the world's population of obese people even as they also have the highest rates of malnourishment. Assumed causes are - as in developed countries - increasing amounts of sugar and junk-food.
The assumption that the world-wide weight increase is a very recent change (since the 1980's is the typical claim) is also being called into question. I found
this excerpt interesting:
There is ample historical evidence that the roots of the obesity pandemic do reach much further back in time than is commonly asserted (Carson 2009, Cuff 1993, Komlos 1987). For the 19th century, we have samples from the West Point Military Academy revealing that by today’s standards BMI values were amazingly low: 19-year-old white cadets had an average BMI value of 20.5, i.e., about the 18th percentile of today’s standards.[1] About 90% of the cadets were below today’s median reference value. In addition, these data indicate that there was very little change in weights in the 19th century. However, another sample from The Citadel military academy in Charleston, SC indicates that a true surge in BMI values took place among those born after the First World War (Figure 1) (Coclanis and Komlos 1995). Note that 18-year-old men increased by some 13 kilograms (28.5 pounds) during the course of the 20th century but half of that increase took place among those born before World War II. Hence, these data indicate that a considerable increase in weight had already taken place by the time the first national survey was taken in 1959-1962 (Figure 1).
Military cadets. Not typically the type of person one would consider a lazy, no-self-control, fat slob, yet their average weight increased by 28 pounds?
Obesity has apparently existed since around 12,000 years ago and the advent of the agricultural revolution. The "very active thrifty genes" you referenced above - which got humans through there hunter/gatherer feast/famine period - creates a problem for many people when food supply is steady.
Shifting gears again, note the "obesity" problem in Samoa. I put it in 'scare quotes' because the situation here calls into question the whole BMI measurement system - another point briefly touched on by someone else earlier. Apparently, given the same lifestyles, Samoans with Polynesian ethnic backgrounds tend to be "fat" compared to those with a Melanesian background. Except, as it turns out, perhaps the Polynesians also have a much higher percentage of muscle (and muscle weighs more than fat). But since BMI is measured by height:weight, it doesn't consider whether the weight is muscle or fat.
And that, again, brings me to the only point I have been making throughout this thread - it simply isn't a matter of telling people to stop eating "so much"