Jarhyn
Wizard
- Joined
- Mar 29, 2010
- Messages
- 15,618
- Gender
- Androgyne; they/them
- Basic Beliefs
- Natural Philosophy, Game Theoretic Ethicist
Yeah, my thought is that the behavior (not to be confused with purpose) of life is "hoard models of all kinds of all structures". Inevitably, one model will be behaviorally homologous to something real which has a survival value for "the hoard", and if that model sees activation, the hoard itself is more likely to remain.That’s basically the speculation about ERVs … the pieces are already there, just waiting for some external event to cause them to be put to use. There is so much geneticperhaps exposure to stress in a part of the body associated with reaching, there can be genetic linkages to activate a genotype for growing taller that was previously methylated and dormant.
material in ERVs, it’s kinda hard to rule it all out. Now that it’s being looked at though, there will likely be some conclusions to be reached soon.
It's a self reinforcing feedback loop about acquiring models for understanding and environment for the purposes of retaining the model.
As a result, anything that makes the hoard of models more helpful and less harmful to its own existence, such as quasi-lamarickian evolution, is going to be heavily selected for.
Very rarely does the hoard get reduced meaningfully, and it's usually when life gets really hard and the cost just of a little bit of DNA in each cell is still meaningfully high. The mammalian genome lost a LOT of pigments, etc..
Darwinian evolution is categorically inferior to any Lamarckian process. The hard part is "worm phenomena", things which use the platform as a substrate for reproduction without contributing to it. In this way, the diversity of cells in a body and their ability to "only mutate a little bit" comes into play in that it allows sacrificial adoption of various messaged adaptations. In short, it has to be limited in some strong way to prevent communicable cancers and other such bullshit.
I suspect we are going to see in the next few years the discovery of a semi-democratic method of demethylation determination on various "risky activations" where some cells will demethylate semi-randomly, some will die, and then the successful demethylations where stress hormones are less expressed than before gets communicated for wide distribution somehow among the body, and a phenotype shift happens.